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Astrocytes initiate inflammation in the injured mouse spinal cord by promoting the entry of neutrophils and inflammatory monocytes in an IL-1 receptor/MyD88-dependent fashion

Since the major quinone forms found in the cells, ubiquinone and plastoquinone, are located into membranes and do not have electrophilic carbon groups that could be substituted by GSH, the question of the GHR physiological substrates is still open