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This review examines the intricate relationship between cigarette smoking, smoking cessation therapies and anti-TB drugs, focusing on the impact of tobacco smoking compounds on liver detoxifying systems, such as influence of polycyclic aromatic hydrocarbons (PAHs) on hepatic cytochrome P450 (CYP450) enzymes mostly, and on metabolism of antituberculous medications

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Furthermore, thyroid hormone could increase biogenesis via activating PGC-1 and promote mitophagy via PINK1 in mice, which helped suppress mitochondria-mediated apoptosis and reversing bleomycin-induced mitochondrial abnormalities in AECs 65

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