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Thus, while NW patients exhibited epigenetic remodeling involving inflammatory and metabolic genes, EBW patients remained epigenetically unresponsive, highlighting nutritional status as a critical determinant of intervention efficacy

(2018) and previous studies implicated increased TrxR1 acetylation as a response to oxidative damage (Banerjee Mustafi et al., 2014), we hypothesized that acTrxR1 may be more active under oxidizing conditions or even resistant to oxidative damage

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