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It has been reported that resistance to daratumumab was associated with the upregulation of complement inhibitory proteins CD55 and CD59 on the MM cells (36)

H2O2 can react with reduced transition metals, via the Fenton reaction, to produce the highly reactive hydroxyl radical (Figure 1, reaction e), a far more damaging molecule to the cell

Hepoxilin A(3) protects beta-cells from apoptosis in contrast to its precursor, 12-hydroperoxyeicosatetraenoic acid

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The target of these exosomes is primarily endothelial cells, which were shown to effectively internalize these bioactive vesicles, subsequently causing an increase in the expression of NF-B [141]