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In this context, ROS-triggered TP53 activation heightens mitochondrial membrane permeability, leading to the release of pro-apoptotic factors from mitochondria

Neuropsychopharmacology 48(5):797805

We have generated the McGill-R-Thy1-APP transgenic (Tg) rat model exhibiting an AD-like amyloid pathology with a prolonged pre-plaque stage, which allows for studying the effects of gradual iA accumulation well before extracellular plaque formation 12,14,36

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Transient induction of antioxidant enzymes and nitric oxide synthase were also delayed or repressed during the regenerative response