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(iii) administration of the imine metabolite NAPQI to human lymphoblastoid cell lines combined with a genome-wide association study (GWAS)

Multivar Behav Res

L., Walker, V

If the intracellular mechanisms of repair of oxidative defects are insufficient or disturbed in turn by the oxidative factors present, there are definitive consequences in some genes or products resulting from the expression of these genes, which causes mutagenesis and modification of the apoptotic mechanism of the cell, thus resulting in the tumor cell ( In the long term, the changes spread and self-sustain with the permanent activation of the autoimmune response and the accumulation of local proinflammatory factors, for example: TNF-alpha, proteases, kinases

Furthermore, we performed additional investigations into the expression of CBS in both normal gastric cells and STAD cells, which unveiled a noteworthy elevation in both CBS mRNA and protein levels in the STAD cell line MKN-45 (Fig