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In a mouse model of alcoholic fatty liver disease, it was demonstrated that the presence of allicin in the diet resulted in a decrease in TNF, IL-1, and IL-6 levels while increasing alcohol dehydrogenase activity (Panyod et al

Surprisingly, despite the extensive literature and the impressive research output over the last 75 years, our understanding of the physiopathological mechanisms underlying diuretic-induced metabolic abnormalities remains notably inadequate

(C) GPX4 expression in mouse livers (Western blot, n = 6 for each group)

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